Nystagmus is a form of involuntary eye movement. It is characterized by alternating smooth puruit in one direction and saccadic movement in the other direction.
Congenital
Acquire Diseases
Case
On 01/04/2010 he was prescribed following medicines. ARGENTUM NIT, AGARICUS, PHYSOSTIGMA, R-78. For one month.
On 27/04/2010 he was prescribed PHOSPHORUS along with above medicines for four months.
Nystagmus is the result of damage to one or more components of the vestibular system, including the semicircular canals, otolith organs, and the vestibulocerebellum.
Pathological nystagmus generally causes a degree of vision impairment, although the severity of such impairment varies widely. Also, many blind people have nystagmus, which is one reason that some wear dark glasses
Variations
- Peripheral nystagmus
- Positional nystagmus
- Gaze Induced nystagmus
- Post rotational nystagmus
- Spontaneous nystagmus
- Central nystagmus.
Causes
The cause for pathological nystagmus may be congenital, idiopathic, or secondary to a pre-existing neurological disorder.
Congenital
Congenital nystagmus occurs more frequently than acquired nystagmus. It can be insular or accompany other disorders (such as micro-ophthalmic anomalies or Down's Syndrome). Congenital nystagmus itself is usually mild and non-progressive. The affected persons are not normally aware of their spontaneous eye movements, but vision can be impaired depending on the severity of the movements.
Types of congenital nystagmus include the following:
- Infantile:
· Albinism
· Aniridia
· Bilateral congenital cataracts
· Optic nerve or macular disease
· Visuo-motor syndrome
Congenital nystagmus is also associated with two X-linked eye diseases known as complete congenital stationary night blindness
Acquire Diseases
Some of the diseases that present nystagmus as a pathological sign:
- Benign Paroxysmal Positional Vertigo
- Head trauma
- Stroke
- Ménière's disease and other balance disorders
- Multiple sclerosis
- Brain tumors (tumors in the posterior fossa.)
- Wernicke-Korsakoff syndrome
- Encephalopathy
- Lateral medullary syndrome
- Optic nerve hypoplasia
- Albinism
- Pelizaeus-Merzbacher disease
- Superior canal dehiscence syndrome
- Tullio phenomenon
Toxic/metabolic
Nystagmus from toxic or metabolic reasons could be the result of the following:
- Alcohol intoxication
- Lithium
- Barbiturates
- SSRIs
- Salicylates
- Benzodiazepines
- Lysergic acid diethylamide
- Phencyclidine
- Ketamine
Central nervous system disorders
- Thalamic hemorrhage
- Tumor
- Stroke
- Trauma
- Multiple sclerosis
- Cerebellar ataxia
Homoeopathic medicines
Agaricus,Calcarea carb, bufo rana, causticum, cicuta v, physo, plut nit, stramonium, zinc met.
Case
This is the case of a man [Mr. S.J.] aged 24 years. He came to see me for nystagmus and albinism. He was under treatment of other system of medicines. He had no relief with that treatment. He came with the severe pain in eyes on reading. He could not open eyes in light and day light. He had involuntary movement of eyes.
On 01/04/2010 he was prescribed following medicines. ARGENTUM NIT, AGARICUS, PHYSOSTIGMA, R-78. For one month.
On 27/04/2010 he was prescribed PHOSPHORUS along with above medicines for four months.
He came on 13/08/2010 with some improvement in pain, photophobia but no improvement in involuntary movements of eyes. He was prescribed PULSATILA with above medicines for four months.
He came on 23/ 12/2010 with improvement in all symptoms. He was repeated same medicines.
On 06/01/2011 patient reported 75% improvement.
On 30/01/2011 patient improved up to 90%. He can read easily without pain in eyes, no photophobia, involuntary movements of eyes also improved. He is enjoying his life. Medicines are continuing for one month.
Dr. G. S. Bhatnagar
D.H.M.S. B.H.M.S.
Research officer
Sewa Mandir
Mobile 9829978284
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